Longer Telomere Length And Slow Of Biological Aging With Regular Physical Exercise Or Workouts
Researchers at Brigham Young University, Provo, Utah, the United States have found that an individual can slow down the biological aging process by almost ten years with a vigorous physical exercise when compared with an individual who is with a sedentary lifestyle (couch potato behavior).
The researchers have conducted a survey among those individuals who participated in the National Health and Nutrition Examination Survey, the United States between the years 1999 - 2002.
About 5,823 people participated in the study. They collected information from the participants such as lifestyle, exercise and demographic information (such as their education, nationality, religion and ethnicity). They also measured the telomere length of the participants.
The researchers have compared the telomere length and the exercise level of the participants.
When compared with individuals with a sedentary lifestyle (or no physical activity), those individuals who are with a high level of physical activity are biologically younger by nine years and those individuals who are with a moderate level of physical activity are biologically younger by seven years.
The researchers say that those individuals who do regular physical exercise or workouts can have a longer telomere length and slow of biological aging (anti-aging). But they are unable to find out a cause and effect relationship.
The author of the study was Professor Larry Tucker, the Brigham Young University and the study was published on April 24, 2017, in the journal Preventive Medicine. Title of the article is "Physical activity and telomere length in U.S. men and women: An NHANES investigation."
The Consumption Of Breast Milk And Omega-3 Fatty Acids Were Linked To The Lower Risk Of Type 1 Diabetes
A study at the National Institute of Health and Welfare, Helsinki, Finland, shows a lower risk of type 1 diabetes (T1D) related autoimmunity in babies with breastfeeding. An earlier study shows that breast milk contains omega-3 (polyunsaturated) fatty acids.
The researchers wanted to find out the association between omega-3 fatty acids and the risk of type 1 diabetes (T1D) in babies.
The researchers have found an inverse association between the risk of type 1 diabetes related to autoimmunity and the consumption of breast milk.
The study also found a decrease in the risk of insulin autoimmunity with higher consumption of cow milk infant formula.
The researchers say that type 1 diabetes (T1D) related autoimmunity in babies is linked with the status of the fatty acid levels in infancy (particularly with the consumption of omega-3 fatty acids). The increased levels of fatty acid are associated with a lower risk of type 1 diabetes (T1D).
A study was conducted on 7,782 babies, aged between 3 and 24 months, who are at genetic risk of type 1 diabetes (T1D).
The blood sample is taken from babies up to the age of 15 years. They checked for islet (in pancreas) autoantibodies in the blood. They collected detailed data regarding breastfeeding exposure and the use of cow milk infant formula.
The study shows that the higher levels of omega-3 fatty acids are associated with a lower risk of early insulin autoimmunity. The study also found that the fatty acid levels of the baby will reflect the type of milk feeding.
The researchers say that the infant can get a protective mechanism against type 1 diabetes (T1D) with the consumption of omega-3 fatty acids. It is important that the mother should take omega-3 fatty acids during breastfeeding.
The author of the study was Dr. Sari Niinisto and the study was published on May 4, 2017, in the journal Diabetologia. Title of the article is "Fatty acid status in infancy is associated with the risk of type 1 diabetes-associated autoimmunity."
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Published by Jammi Vasista, Chennai, India.