An Association Between The Frozen Shoulder And Type 2 Diabetes
A study at the Hadassah-Hebrew University Medical Center, Jerusalem, Israel shows that the frozen shoulder (or painful shoulder) could be a warning sign of type 2 diabetes (T2D). Joint pain or persistent stiffness in the joint are the symptoms of the frozen shoulder. The normal shoulder movements will become difficult with the frozen shoulder, affecting the daily activities.
The frozen shoulder is also known as the shoulder contracture or the adhesive capsulitis. This problem generally occurs in people who underwent shoulder surgery. An earlier study shows that the shoulder disorders are affecting between 10 and 22 percent of the patients with type 2 diabetes (T2D) compared between two and four percent in the patients without type 2 diabetes.
Experts believe that the hyperglycemia (high blood sugar levels) can increase the swelling and inflammation in the joints leading to the frozen shoulder. The study was published July 11, 2017, in the Orthopaedic Journal of Sports Medicine. Title of the article was "Should Patients With Frozen Shoulder Be Screened for Diabetes Mellitus?".
Frozen shoulder: Restricted movement of the shoulder associated with pain and stiffness is called frozen shoulder. The frozen shoulder may be due to the overuse of the shoulder, shoulder injury or diseases such as a stroke or diabetes.
This pain comes in and goes out slowly over a period. This can be prevented if the pain is due to injury. The following are the common risk factors for the frozen shoulder.
An individual suffering from frozen shoulder should consult a doctor to know about the exercise programs to prevent chronic shoulder stiffness. The American Association of Orthopaedic Surgeons (AAOS) suggests a frequent and gentle crossover arm stretch exercise to get the relief from frozen shoulder.
Altering The Sense Of Smell Can Help Weight Loss And Type 2 Diabetes
A study on mouse models at the University of California shows a lower risk of type 2 diabetes (T2D) and weight loss by losing the sense of smell (a key to enjoy the eating). The gene therapy technique was used in the experiment to find out the benefits of losing the sense of smell in the mouse models.
The researchers have observed an increase in the burning of fat and calories in the obese mouse models with high blood sugar (glucose) levels, but without the sense of smell. But the mouse models have regained the weight and high sugar levels after regaining the sense of smell. The researchers have observed an increase in the noradrenaline hormone with the loss of sense of smell. Noradrenaline hormone is a risk factor for a heart attack.
The researchers have suggested that the burning of excess fat and calories in the people who cannot smell the food instead of storing calories in the body (in the form of fat). This study indicates an association between the body metabolism and the smell system (part of the sensor system. Olfactory). The researchers say that this study suggests the role of the sensory neurons involved in the smell system of the body metabolism. This can help an individual with a difficulty in losing weight.
The authors say that a drug can be made to block the genes (and part of the sensor system) involved in the sense of smell. This study provides a practical and possible solution for the obese patient with type 2 diabetes and difficulty in losing weight.
The senior author of the study was Dr. Andrew George Dillin, professor in molecular and cell biology and an investigator at Howard Hughes Medical Institute, Maryland, United States. The study was published on July 5, 2017, in the journal Cell Metabolism. Title of the article was "The Sense of Smell Impacts Metabolic Health and Obesity".
The Diabetes News Chronicle does not provide medical advice, diagnosis or treatment. Information in Diabetes News Chronicle is to support and not to replace medical advice given by the surgeon or physician or doctor. The published article is not a medical advice by the OWNER of the "Diabetes News Chronicle" website or by the AUTHOR of the article.
Published by Jammi Vasista, Chennai, India.